4 edition of Heart perfusion, energetics and ischemia found in the catalog.
1983 by Plenum in cooperation with NATO Scientific Affairs Division in New York, London .
Written in English
Includes bibliographies and index.
|Statement||(proceedings of a NATO Advanced ResearchWorkshop on Microvascular, Rheological, Metabolic and Heat-Transfer Aspects of the Heart , Relation to Ischemia and Thrombosis, held July 4-11, 1982, in Château de Bonas, Bonas, France) ; edited by Leopold Dintenfass, Desmond G. Julian and Geoffrey V.F. Seaman.|
|Series||NATO advanced science institute series. Series A, Life sciences -- v.62, NATO advanced study institute series -- v.62.|
|Contributions||Dintenfass, Leopold., Julian, Desmond G., Seaman, Geoffrey V. F., NATO Scientific Affairs Division.|
|The Physical Object|
|Number of Pages||707|
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One of the purposes of the Convenor was to relate clinical haemorheology to the heart energetics and heart metabolism. This was only partly established, as difficulties of communications between different fields, difficulties of semantics 3/5(1). One of the purposes of the Convenor was to relate clinical haemorheology to the heart energetics and heart metabolism.
This was only partly established, as difficulties of communications between different fields, difficulties of semantics and of specialized out looks could not be overcome within a. The Paperback of the Heart Perfusion, Energetics, and Ischemia by Leopold Dintenfass at Barnes & Noble. FREE Shipping on $35 or more.
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Heart perfusion, energetics, and ischemia. New York: Plenum Press, © (OCoLC) Online version: NATO Advanced Research Workshop on Microvascular, Rheological, Metabolic, and Heat-Transfer Aspects of the Heart, Relation to Ischemia and Thrombosis ( Bonas, France).
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The Human Heart as a Pump.- The Heart as a Muscle and a Pump: Ventricular Function of the Heart in Ischemic Heart Disease.- Control of Cardiac Output in Sustained Essential Hypertension: A Biomathematical Approach.- Discussion The Heart as a Muscle and a Pump.- 3: Microcirculation and Myocardial Perfusion.- The Coronary Arteries The Heart as a Muscle and a Pump: Ventricular Function of the Heart in Ischemic Heart Disease.- Control of Cardiac Output in Sustained Essential Hypertension: A Biomathematical Approach.- Discussion The Heart as a Muscle and a Pump.- 3: Microcirculation and Myocardial Perfusion In the normal heart, the abrupt onset of ischemia leaves little time for the heart to adapt and thus, the relation between flow and function is completely a reflection of acute intrinsic physiological adaptations to by: The human heart is an unusual and efficiently designed pump.
Its main function is to pump blood through the entire circulation to meet the hematologic requirements of all cells of the body. This pump is most unusual in that the source of energy for the pump is located in the walls of the pump by: 1. When arteries are narrowed, less blood and oxygen reaches the heart muscle.
This is also called coronary artery disease and coronary heart disease. This can ultimately lead to heart attack. Ischemia often causes chest pain or discomfort known as angina pectoris.
Recently cardiac imaging has played an important role in the detection of ischemia. Perfusion abnormalities can be detected by single-photon emission computer tomography (SPECT) myocardial perfusion imaging (MPI) and mechanical dysfunction can be detected by echocardiography or gated by: 3.
After 5 minutes, sodium pentobarbital ( mg/kg) I.P. is given. The heart is rapidly excised (with lungs and thymus intact) and arrested in energetics and ischemia book cold KH buffer. While kept on ice, the lungs are quickly removed. The lobes of the thymus are identified and gently peeled back to expose the by: In energetics and ischemia book three groups, the isolated rabbit hearts were subjected to 30 minutes of hypoperfusion followed by 60 minutes of reperfusion.
In groups 2 energetics and ischemia book 3, the hearts underwent the ischemic preconditioning procedure (3 minutes of no-flow ischemia followed by 8 minutes of Cited by: 2.
CARDIOVASCULAR PHARMACOLOGY administration. During intravenous therapy with NTG, if blood pressure (BP) drops and ischemia is not relieved, the addition of phenylephrine will allow coronary perfusion pressure (CPP) to be maintained while allowing higher doses of NTG to be used for ischemia.
Energetics of Ca 2+ homeostasis during ischemia–reperfusion on neonatal rat hearts under high-[K +] cardioplegia Alicia E. Consolini, Patricia Bonazzola» AbstractCited by: Heart perfusion. In heparinized (50 U ip) and anesthetized (75 mg/kg pentobarbital sodium ip) wild-type and AK1 knockout mice (40–50 g), hearts were excised and retrogradely perfused with a 95% O % CO 2-saturated Krebs-Henseleit (K-H) solution (in mM: NaCl, KCl, CaCl 2, 19 NaHCO 3, MgSO 4, glucose, EDTA; 37°C) at a perfusion pressure of 70 by: Cardiac ischemia happens when an artery becomes narrowed or blocked for a short time, preventing oxygen-rich blood from reaching the heart.
In most cases, a temporary blood shortage to the heart causes the pain of angina pectoris. But in other cases, there is no pain. These cases are called silent ischemia. Shaw LJ, Weintraub WS, Maron DJ, et al.
Baseline stress myocardial perfusion imaging results and outcomes in patients with stable ischemic heart disease randomized to optimal medical therapy with or without percutaneous coronary intervention.
Am Heart J ; Decreasing coronary perfusion causes an immediate decrease in contractile function via unknown mechanisms. It has long been suspected that this contractile dysfunction is caused by ischemia-induced changes in cardiac by: 8. Ischemia or ischaemia is a restriction in blood supply to tissues, causing a shortage of oxygen that is needed for cellular metabolism (to keep tissue alive).
Ischemia is generally caused by problems with blood vessels, with resultant damage to or dysfunction of ciation: /ɪˈskiːmiə/. The irrreversibile ischemic heart disease is a term too vague because you should specify whether directed specifically to the cell or to a greater or lesser range of myocardial injury employee on.
Ischemic heart disease remains a major factor of death and disability worldwide , and the mitochondrial Ca 2+ mishandling in cardiomyocytes during ischemia/reperfusion injury is well.
Effects of pyruvate on the energetics of rat ventricles stunned by ischemia–reperfusion. Patricia Bonazzola, a María Inés Ragone, b Alicia E. Consolini b. a Cátedra de Biofísica, Facultad de Odontología e Instituto de Investigaciones Cardiológicas (CONICET, Facultad de Medicina), Universidad de Buenos Aires (UBA), by: 5.
The initial ischemia injury produced by occlusion of the coronary circulation can be minimized in MI patients by the use of angioplasty to restore perfusion in a timely fashion. While this intervention has greatly reduced the number of deaths due to acute MI, restoration of blood flow into the ischemic area results in I/R injury that leads to Cited by: atp-sensitive k + (K ATP) channels, which are highly expressed in myocardial sarcolemma, serve as membrane metabolic sensors that translate fluctuations in cellular energetics into regulation of electrical activity (1, 24, 25, 40).Nucleotide-dependent K + permeation through Kir, the inwardly rectifying pore-forming core of the K ATP channel, is gated by ATPase activity of the regulatory Cited by: Supply.
The heart normally receives 4% of cardiac output, or ~ mL/min of acids and lactate are the predominant sources of energy, although glucose can be utilized (depending on glucose and insulin concentrations, as well as PaO2).
Demand. Impact of PVA on mVO2 (Image by Thiele, Nemergut, Lynch)Normal mVO2 is mL/g/min. mVO2 is related to the area inside the pressure. Alternatively, in a starkly opposite manner, the hypotension of slow-wave sleep may lead to malperfusion of the myocardium because of reduced coronary perfusion pressure through stenotic vessel segments (Fig.
44 Several investigat45,46 have attributed nocturnal MI and myocardial ischemia to the relative hypotension of NREM sleep, which “reduces the volume and velocity of blood. Start studying Perfusion: Heart Failure. Learn vocabulary, terms, and more with flashcards, games, and other study tools.
Search. -ischemic heart disease-cardiomyopathy-infections. Systolic Dysfunction -tissue perfusion is adequate for demand.
Decreasing coronary perfusion causes an immediate decrease in contractile function via unknown mechanisms. It has long been suspected that this contractile dysfunction is caused by ischemia. Remote ischemic conditioning is applied to a heart-distant organ, in this case the upper extremity through blood pressure cuff in- and deflations (row four).
The stimulus can be applied prior to the heart (main) ischemia or—in case of perconditioning (row five)—during the main by: 2.
INTRODUCTION. Ischemia occurs when blood flow to the myocardium is reduced ().Ischemia of prolonged duration induces myocardial infarction (MI), and MI is a common cause of heart failure ().Ischemic cardiomyopathy is the most common cause of heart failure and can arise from remodeling after an acute ST segment elevation myocardial infarction (STEMI) from multiple small nontransmural Cited by: Myocardial perfusion Gated SPECT study is positive for medium size fixed perfusion defect involving the adjacent areas of inferior wall and infero.
Myocardial. Medium perfusion defect moderate intensity in basal mid inferior basal inferior partial reversibility. Ef54%. TID. 97 abnormal redegenson. CMR can identify ischemia by assessing perfusion and wall motion with stress.
CMR has high accuracy and excellent prognostic data in the evaluation of ischemia. Fractional flow reserve derived from CCT is promising for evaluation of ischemia, but requires further study. CMR has become a gold standard for imaging of myocardial infarction and. Mechanisms Underlying Acute Protection From Cardiac Ischemia-Reperfusion Injury ELIZABETH MURPHY AND CHARLES STEENBERGEN Vascular Medicine Branch, National Heart, Lung, and Blood Institute, National Institutes of Health, Department of Health and Human Services, Bethesda; and Johns Hopkins Medical Institute, Baltimore, Maryland I.
Introduction. mic heart disease (IHD) (angina, myo-cardialinfarction,cardiacsuddendeath, and heart failure) are attributed to the effects of epicardial coronary obstruc-tive lesions. In this view, stenotic or occluded coronary arteries hamper downstreambloodﬂow,reducemyocar-dial perfusion, and cause contractile dysfunction.
Accordingly, in patients. Exercise-induced protection against reperfusion arrhythmia involves stabilization of mitochondrial energetics. Rick J. Alleman, Alvin M. Tsang Two-photon microscopy whole heart imaging during ischemia-reperfusion.
Myocytes were loaded on a perfusion chamber housed on the confocal microscope stage and enclosed in glass to minimize O 2 Cited by: Myocardial infarction (MI) or acute myocardial infarction (AMI) is the death of heart muscle from the sudden blockage of a coronary artery by a blood clot.
Myocardial infarction most commonly due to occlusion (blockage) of a coronary artery following the rupture of a vulnerable atherosclerotic plaque, which is an unstable collection of lipids (cholesterol and fatty acids) and white blood cells Missing: energetics and ischemia.
Tio RA, Dabeshlim A, Siebelink H-MJ, et al. Comparison between the prognostic value of left ventricular function and myocardial perfusion reserve in patients with ischemic heart disease.
J Nucl Med. ; – Heart perfusion. All animal experiments were approved by Institutional Animal Care and Use Committee and conformed to the National Institutes of Health Guide for the Care and Use of Laboratory Animals [DHHS Publication No.
(NIH)RevisedOffice of Science and Health Reports, Bethesda, MD ]. Fed, adult male Sprague-Dawley rats were anesthetized with ketamine ( mg/kg ip) and Cited by: Overview of Ischemic Heart Disease Myocardial ischemia is caused by an imbalance between myocardial oxygen supply and demand: major symptom is angina pectoris: uncomfortable pain and sensation in the chest or nearby anatomical structures produced by myocardial ischemia.
Decreasing coronary perfusion causes an immediate decrease in contractile function (EF)via unknown mechanisms. It has long been suspected that this contractile dysfunction is caused by ischemia-induced changes in cardiac energetics (physics of energy and its transformation)." Your husband's condition may be due to sepsis.Isolated left ventricular non-compaction is a rare congenital cardiomyopathy.
Patients frequently present with signs of heart failure and dyspnea on exertion. Myocardial perfusion tests using single photon emission computed tomography are frequently used for evaluation of ischemia. Left ventricular (LV) hypertrophy in aortic stenosis (AS) is characterized by reduced myocardial perfusion reserve due to coronary microvascular dysfunction.
However, whether this hypoperfusion leads to tissue deoxygenation is unknown. We aimed to assess myocardial oxygenation in severe AS without obstructive coronary artery disease, and to investigate its association with Cited by: